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This is an interesting point bc on Amboss it says the mechanism is actually a compensatory immune repsonse where the body forms immune complexes to activate the platelets. This inadvertently causes them to clump together, hypercoagulate, and reduce free platelet counts. So it is not a direct thrombocytopenia. Likewise the TTP from ADP receptro blockers is also indirect. I have tried to integrate these points in the suggestion.