No, the original card was correct. Amboss clearly says ascending thoracic = CMN, descending thoracic = atherosclerosis.
Here is Amboss on thoracic aneurysms:
“Pathophysiology
Ascending thoracic aortic aneurysm: most often due to cystic medial necrosis
Descending thoracic aortic aneurysm: typically a result of atherosclerosis
Inflammation and proteolytic degeneration of connective tissue proteins (e.g., collagen and elastin) and/or smooth muscle cells in high-risk patients → loss of structural integrity of the aortic wall → widening of the vessel
The aneurysmatic dilatation of the vessel wall may cause disruption of the laminar blood flow and turbulence.
Possible formation of thrombi in the aneurysm → peripheral thromboembolism”
And Amboss on abdominal aneurysms:
“Etiology
Risk factors [2]
Smoking (most important risk factor)
Advanced age
Atherosclerosis (ASCVD)
Hypercholesterolemia and arterial hypertension
Positive family history
Male sex
Trauma”
“Pathophysiology
Inflammation and proteolytic degeneration of connective tissue proteins (e.g., collagen and elastin and/or smooth muscle cells) in high-risk patients → loss of structural integrity of the aortic wall → widening of the vessel → mechanical stress (e.g., high blood pressure) acts on weakened wall tissue → dilation and rupture may occur.
The aneurysmatic dilatation of the vessel wall may cause disruption of the laminar blood flow and turbulence.
Possible formation of thrombi in the aneurysm → peripheral thromboembolism”